FAQ's

Liver Healing Formula (LHF) is a first of its kind product derived from multiple combinations of herbs
with proven results for Liver Diseases. It is scientifically processed without affecting the chemical
properties of the active ingredients (coumestans, alkaloids, thiopenes, flavonoids, polyacetylenes,
triterpenes and their glycosides). Extracts and metabolites from these Herbal parts have been known to
possess pharmacological properties to give maximum benefits.
Liver Healing Formula (LHF) has been well researched and tried and also approved by AYUSH, Govt. of
India.

Liver Healing Formula (LHF) has been taken along with other prescription and non-prescription
medications. There have been no reported side-effects. In the event of any drug toxicity or side-effects,
the liver would be the primary organ to be affected, resulting in elevated liver enzymes. Since Liver
Healing Formula normalizes elevated liver enzymes, it is very unlikely that such a product can have any
side-effects.

In most cases recovery can be felt within 3-5 days of using Liver Healing Formula.

Yes. In fact a much drastic reduction in the viral count has been seen, without going through the much
severer side-effects of Interferon usage.

Liver Healing Formula can be taken with most commonly prescribed medications (anti-diabetic, anti-
hypertensive, antacids, analgesics, anti-inflammatory, food supplements, etc) and no contradictory
reports have been received whatsoever. However, drug interactions may vary from person to person.
Individuals with other underlying health conditions who are on different medications have to seek
medical advice from their physicians.

Yes, Liver Healing Formula can be taken as a preventive medicine. It has shown to be helpful to
individuals who consume alcohol and also to those who are Hepatitis B or C positive but are not showing
any symptoms.

The duration of the treatment varies from case to case depending on the severity of the disease and the
individual. Commonly, if the elevated liver enzymes are back to normal, one can stop taking Liver
Healing Formula.
In case of Hepatitis B or C once the elevated liver enzymes drop to normal with significant reduction in
viral load the dosage may be reduced to 1 or 2 times a day. In case of further recovery the dose may be
further reduced to 1 time every alternate day. If subsequent medical tests indicate progression of the
disease the dosage should be increased.
After initial recovery, if Liver Healing Formula is taken on alternate days, it is very unlikely that the
disease will progress. In individuals who clear the virus and their blood tests show viral load as not
detectable, there is no need to continue Liver Healing Formula.

Yes. When the liver is not functioning normally, levels of Albumin, a protein synthesized in the liver, are
also reduced. Low Albumin results in the decreased kidney filtration rate and in such cases Albumin has
to be administered intravenously. Liver Healing Formula increases Albumin production naturally and
hence helps restore normal kidney filtration.

The liver and how it works?

The liver is the largest organ in the body weighing about 3 pounds and is the largest solid organ in the
body and plays an important role, performing many difficult functions that are essential for life. Your
liver serves as your body’s internal chemical power plant. It is impossible to live without the liver and the
health of the liver is a major factor in the quality of one’s life. Some important functions of the liver are:

* Manufacturing blood proteins that aid in clotting, oxygen transport, and immune system
function.
* To convert the food we eat into stored energy, in the form of glycogen, and chemicals necessary
for life and growth.
* To break down saturated fat and producing cholesterol.
* To act as a filter to remove alcohol and toxic substances from the blood and convert them to
substances that can be excreted from the body;
* To process drugs and medications absorbed from the digestive system, enabling the body to use
them effectively and ultimately dispose of them;
* To create and export important chemicals used by the body.

The most important thing to recognize about liver disease is that up to 50 percent of individuals with
underlying liver disease have no symptoms. The most common symptoms are very non-specific and they
include fatigue or excessive tiredness, lack of drive, occasionally itching. Signs of liver disease that are
more prominent are jaundice or yellowing of the eyes and skin, dark urine, very pale or light colored
stool or bowel movements, bleeding from the GI tract, mental confusion, and retention of fluids in the
abdomen or belly.

The liver is a unique organ. It is the only organ in the body that is able to regenerate… that is completely
repair the damage. With most organs, such as the heart, the damaged tissue is replaced with scar, like
on the skin. The liver, however, is able to replace damaged tissue with new cells. An extreme example is
a patient who suffers an overdose from Tylenol. In this example up to 50 – 60 percent of the liver cells
may be killed within 3 – 4 days. However, if no other complications arise, the patient’s liver will repair
completely, and a liver biopsy after 30 days will appear completely normal with no signs of damage and
no scar. However, the long-term complications of liver disease occur when regeneration is either
incomplete or prevented by progressive development of scar tissue within the liver. This occurs when
the damaging agent such as a virus, a drug, alcohol, etc., continues to attack the liver and prevents
complete regeneration. Once scar tissue has developed it is very difficult to reverse that process. Severe

scarring of the liver is the condition known as cirrhosis. The development of cirrhosis indicates late stage
liver disease and is usually followed by the onset of complications.

Blood tests are useful in the determination of ALD. Blood tests can establish alcohol as the cause of the
disease but cannot determine the seriousness of the illness. Aminotransferase abnormalities are usually
indicative of ALD. These are AST, ALT, and AST/ALT ratios. There is increased serum activity of gamma
glutamyl transpeptidase (GGTP) in chronic alcohol users. Sixty percent of alcoholics have elevated GGTP
in combination with elevated AST levels. Serum electrolytes, mean corpuscular volume (MCV) and
serum uric acid levels are also indicative of ALD in combination with results from other tests.
Prothrombin time (ability of the blood to clot) may be indicative of mild hepatic disease, but may be
present in hepatic failure.
Liver biopsy is not always necessary to confirm the diagnosis of ALD, but is the most sensitive measure
of disease stage and is useful in predicting the disease course.
Abdominal ultrasound and CT scanning may also be performed. Abdominal ultrasound is useful in the
assessment of fatty content of the liver. CT scanning detects cirrhosis, portal hypertension and tumors.

Diet And Nutrition

Nutrition and the liver are connected in many ways. Since most of what we eat, breathe, and absorb
through our skin must be refined and detoxified by the liver, special attention to nutrition and diet can
help keep the liver healthy. Approximately 85–90 percent of the blood that leaves the stomach and
intestines carries important nutrients to the liver where they are converted into substances the body
can use.

Poor nutrition is not generally a cause, with the exception of alcoholic liver disease, and liver disease
found among starving populations. It is much more likely that poor nutrition is the result of chronic liver
disease, and not the cause. Good nutrition with a balanced diet can actually help the damaged liver
regenerate new liver cells. In fact, in some liver diseases, nutrition becomes an essential form of
treatment. Ask your doctor.Poor nutrition is not generally a cause, with the exception of alcoholic liver disease, and liver disease
found among starving populations. It is much more likely that poor nutrition is the result of chronic liver
disease, and not the cause. Good nutrition with a balanced diet can actually help the damaged liver
regenerate new liver cells. In fact, in some liver diseases, nutrition becomes an essential form of
treatment. Ask your doctor.

High protein diets are generally well-tolerated by healthy adults. But a dramatic increase in protein-rich
foods may be dangerous for people with severe liver or kidney disease for the following two reasons:
 Such individuals may lack the ability to get rid of the waste products left from protein
metabolism.

 These diets may also limit intake of other important foods, such as fruits, vegetables, and whole
grains.
You may not know if you have liver disease. For this and other reasons, ask your doctor before starting
any diet.

Many chronic liver diseases are associated with malnutrition. The most common is cirrhosis, which is the
scarring of the liver. Cirrhosis can occur by repeated injury to liver cells, which can be caused by
excessive alcohol intake, chronic viral hepatitis, exposure to certain drugs or toxic substances, and a
variety of other causes.
People with cirrhosis often experience loss of appetite, nausea, vomiting, and weight loss, giving them a
thin appearance. Diet alone does not contribute to the development of this liver disease generally,
although non-alcoholic fatty liver, associated with obesity, can lead to cirrhosis. People who are well-
nourished but drink large amounts of alcohol are also at risk for alcoholic disease.

Fatty liver is an abnormal accumulation of fat (small or large droplets) in the cytoplasm of liver cells and
may often be accompanied by fibrosis. Fatty liver is present in approximately 90-100% of heavy drinkers
(who consume more than 80 grams of alcohol per day over a 5 year period). Palpable liver enlargement
is also characteristic of fatty liver and is present in about 90%of these patients. Conditions other than
heavy alcohol consumption may also cause fatty liver.
Fatty Liver Disease, also known as "fatty infiltration of the liver," is not caused by excessive eating of
fats, although obesity is a risk factor for fatty liver. In some patients, the fat is associated with
inflammation and scarring and may lead to cirrhosis. Nutritional causes of fat in the liver include alcohol,
starvation, obesity, protein malnutrition, and intestinal bypass operation for obesity. Diabetes is another
factor associated with fatty liver.

Chemicals And Drugs

Yes, chemicals can cause chronic liver disease and cirrhosis. Usually, chronic liver disease develops only
after long-term use of a drug. Excessive exposure to certain drugs and chemicals may cause tumors of
the liver as well.

Yes, liver damage is common in people who are regular illegal drug users. Most instances of liver
damage in these individuals result from viral hepatitis caused by sharing contaminated needles and
using alcohol.

Alcohol

Yes, but it is only one of the many causes, and the risk depends on how much you drink and over how
long a period. Some causes include viruses, hereditary defects, and reactions to drugs and chemicals.

First, it should be understood that alcohol is a poison. Any amount of alcohol can produce damage to
the liver. In an otherwise healthy person with no underlying liver problems, the general rule of thumb is
different for men and women. Men metabolize and are able to clear alcohol more efficiently than
women due to body size, body fat and certain enzymes. Because of this the maximum "safe" daily intake
of alcohol for a woman is 1 – 2 drink per day; for the male it is 3 – 4 over a 24-hour period. It is also
important to recognize the body and the liver in particular does not distinguish between different forms
of alcohol. Beer and wine are not "safer" than whiskey or spirits. One drink is defined as one shot (1 and
1/4 ounces) of whiskey or spirits, one four-ounce can of wine or one 12-ounce can of beer. If an
individual has an underlying liver condition such as hepatitis B or C, or prior damage from alcohol or
other diseases, the liver is very sensitive to any amount of alcohol. In those conditions, the only safe
dose of alcohol is zero.

Yes. Even moderate amounts of alcohol can have toxic effects when taken with over-the-counter drugs
containing acetaminophen. If you are taking over-the-counter drugs, be especially careful about drinking
and don't use an alcoholic beverage to take your medication. Ask your doctor about precautions for
prescription drugs.

Alcoholic hepatitis is an inflammation of the liver that lasts one to two weeks. Symptoms include loss of
appetite, nausea, vomiting, abdominal pain and tenderness, fever, jaundice, and sometimes, mental
confusion. It is believed to lead to alcoholic cirrhosis over a period of years. Cirrhosis involves
permanent damage to the liver cells. "Fatty Liver" is the earliest stage of alcoholic liver disease. If the
patient stops drinking at this point, the liver can heal itself.

Yes. Alcoholic hepatitis is mostly found in alcoholics, but it also occurs in people who are not alcoholics.

No. Some alcoholics may suffer seriously from the many physical and psychological symptoms of
alcoholism, but escape serious liver damage. Alcoholic cirrhosis is found among alcoholics about 10–25
percent of the time. If alcoholic hepatitis is detected and treated early, cirrhosis can be prevented. However, if untreated for a long period of time, it may be fatal, especially if the patient has had previous
liver damage.

Cirrhosis is the scarring of the liver caused by long-term alcohol abuse or chronic viral hepatitis. In
children, the most frequent causes are biliary atresia, which often requires a liver transplant.

Complications of ALD are usually caused by the systemic complications of hepatic injury. These include:
portal hypertension, an obstruction of the normal blood flow through the liver and reduction in
functional hepatocyte mass. These conditions may occur in those with alcoholic hepatitis or alcoholic
cirrhosis and in other non-alcohol related liver diseases.
Portal hypertension results in an elevation in pressure throughout the vascular tree above the portal
vein. This elevation in pressure may cause the formation of ascites (accumulation of serous fluid in the
abdomen) and increased blood flow through alternative pathways resulting in the development of
varices and hypersplenism. Spontaneous bacterial peritonitis may occur in the acutely ill cirrhotic
patient.
Reduction in functional liver mass causes hepatic encephalopathy (mental dysfunction caused by an
accumulation of nitrogenous wastes in the blood and brain), coagulopathy (dysfunctional clotting
mechanism due to decreased synthesis of clotting factors) and hypoalbuminemia (decreased synthesis
and hepatic secretion of albumin, a protein responsible for maintaining serum osmotic pressure). When
albumin content is decreased, the water tends to move out of serum into the tissues (edema or
swelling) or into the peritoneal space (area surrounding organs in the abdomen) or ascites.

Nonalcoholic steatohepatitis (NASH) is characterized by histopathological features of alcoholic hepatitis
in patients drinking insignificant amounts of alcohol. Up to 20 per cent of patients with elevations of
transaminases of unknown significance have been reported to have NASH. In about 80 per cent of cases
NASH is associated with obesity and type 2 diabetes mellitus, especially in middle aged women. Hepatic
steatosis, hyperinsulinemia, endotoxins, proinflammatory cytokines, oxidative stress and genetic factors
are pathogenetically important. In its strict form NASH is characterized by steatosis, signs of
hepatocellular injury, necroinflammation and fibrosis. Most patients are asymptomatic or have mild,
noncharacteristic right upper quadrant complaints. Transaminase values are below 100 U/l, alanine
aminotransferase is higher than aspartate aminotransferase. In most patients long term prognosis is
good, however, in some patients NASH is progressive and may lead to cirrhosis. Management includes
careful weight reduction and treatment of diabetes. Therapy with ursodeoxycholic acid, vitamin E and
insulin sensitizing agents has yielded promising initial results.

Viral Hepatitis

Viruses have either RNA (Hepatitis C) or DNA (Hepatitis B), and being non-living they are referred to as
viral particles. They multiply within the host’s cell by using the host cells’ DNA or RNA. As the number of
virus particles / virions increase inside the liver cell, the infected Liver Cells rupture and each Virion that
comes out is capable of invading another healthy Liver Cell. This rapid viral multiplication causes the
death of millions of healthy Liver Cells.

There are various other viruses known to cause liver disease: hepatitis A, B, C, D, and E, which vary in
their severity and characteristics.
Hepatitis C can lead to serious, permanent liver damage and, in many cases, death.
Hepatitis A: Is a liver disease caused by the hepatitis A virus (HAV). Hepatitis A can affect anyone and
can occur in situations ranging from isolated cases of disease to widespread epidemics due to
contaminated food or water.
Hepatitis B: Is a serious disease caused by a virus that attacks the liver. The virus, which is called the
hepatitis B virus (HBV), can cause lifelong infection, cirrhosis (scarring) of the liver, liver cancer, liver
failure, and death. HBV is transmitted by body fluids and blood.
Hepatitis C: Is a liver disease caused by the hepatitis C virus (HCV), which is found in the blood of
persons who have the disease. HCV is spread by contact with the blood of an infected person.
Hepatitis D: Is a liver disease caused by the hepatitis D virus (HDV), a defective virus that needs the
hepatitis B virus to exist. (HDV) is found in the blood of persons infected with the virus.
Hepatitis E: Is a liver disease caused by the hepatitis E virus (HEV) transmitted in much the same way as
the hepatitis A virus. Hepatitis E, however, does not occur often.

Hepatitis is a generic term. It indicates inflammation and damage to liver cells. This damage can be
caused by drugs, toxins, alcohol, inherited diseases, certain metabolic diseases and viruses. Commonly,
however, hepatitis refers to viral hepatitis. There are a wide variety of viruses that can cause hepatitis,
but again most commonly the term refers to the viruses designated A, B, C, D, E, and G. In the World,
the most common causes are hepatitis A, B, and C.

There are vaccines to prevent hepatitis A and B. Hepatitis B is a disease that could be completely
eradicated with universal vaccination. It is now one part of the newborn vaccination series. Attempts are
ongoing to vaccinate all children by the time they reach junior high age. Adults who are in high-risk
occupations such as the health care field or carry out high-risk activities, such as IV drug use and
multiple sexual partners should also be vaccinated. Hepatitis A vaccine is recommended in a number of
child-care settings and should be discussed with your pediatrician. Adults or children traveling to areas

of the world where hepatitis A is very common, including all underdeveloped or poorly developed
countries, should be vaccinated before they go. Any individual with underlying chronic liver disease that
is not due to hepatitis B, particular those with hepatitis C or cirrhosis should be vaccinated against both
Hepatitis A and Hepatitis B, unless they are already immune.

Liver Transplant

Yes and no. If medical treatment is likely to allow prolonged survival with good quality of life,
transplantation would be reserved for the future. However, ideally the surgery is undertaken before the
terminal stage of the disease when the person is too ill to withstand major surgery and will not survive
until a suitable donor is available.

Before surgery, the risks are mainly the development of some acute complication of the disease which
might render the patient unacceptable for surgery. With transplantation there are risks common to all
forms of major surgery, as well as technical difficulties in removing the diseased liver and implanting the
donor liver. One of the major risks for the patient is not having any liver function for a brief period.
Immediately after surgery, bleeding, poor function of the grafted liver, and infections are major risks.
The patient is carefully monitored for several weeks for signs of rejection of the liver.